Diagnosis and treatment of Enhanced Myometrial Vascularity: Filling in the Knowledge-Gap

Two kinds of AVMs, shortcuts between arteries and veins of organs bypassing the capillaries. are known: Congenital AVM not associated with pregnancies and acquired AVM, develops related to pregnancy. They represent a faulty, incomplete and prolonged regression of the physiologically remodeled, dilated uterine spiral arteries supplying the placenta through a high velocity, low resistance vascular network. They test the anxious caretakers as well as patients. Unfortunately, on US, at times the slow regression turns into a threatening, persistent form expressed by heavy vaginal bleeding. Van Schoubroeck et al.2 initiated the change of terminology of uterine AVMs in the backdrop of pregnancy. To cement the new term: EMV, Dewide et al.3) calls to relinquish the misused term of AVM and adopt the pathophysiological as well clinically correct term of EMV by declaring: ‘Time has come to stop misusing the term arterio-venous malformation!’ differentiating AVM from EMV.